Acta Neuropathologica

Papers
(The H4-Index of Acta Neuropathologica is 48. The table below lists those papers that are above that threshold based on CrossRef citation counts [max. 250 papers]. The publications cover those that have been published in the past four years, i.e., from 2020-05-01 to 2024-05-01.)
ArticleCitations
Neuropathology of COVID-19: a spectrum of vascular and acute disseminated encephalomyelitis (ADEM)-like pathology384
Plasma p-tau231: a new biomarker for incipient Alzheimer’s disease pathology292
The physiological roles of tau and Aβ: implications for Alzheimer’s disease pathology and therapeutics225
Distribution patterns of tau pathology in progressive supranuclear palsy219
Plasma p-tau181 accurately predicts Alzheimer’s disease pathology at least 8 years prior to post-mortem and improves the clinical characterisation of cognitive decline212
TREM2 activation on microglia promotes myelin debris clearance and remyelination in a model of multiple sclerosis185
Distinct amyloid-β and tau-associated microglia profiles in Alzheimer’s disease167
Cerebral blood flow decrease as an early pathological mechanism in Alzheimer's disease154
Periphery and brain, innate and adaptive immunity in Parkinson’s disease140
CDKN2A/B homozygous deletion is associated with early recurrence in meningiomas120
APOE and TREM2 regulate amyloid-responsive microglia in Alzheimer’s disease118
Correlates of critical illness-related encephalopathy predominate postmortem COVID-19 neuropathology114
Accumulation of amyloid precursor protein C-terminal fragments triggers mitochondrial structure, function, and mitophagy defects in Alzheimer’s disease models and human brains113
Neuropathological consensus criteria for the evaluation of Lewy pathology in post-mortem brains: a multi-centre study109
Cryo-EM structures of tau filaments from Alzheimer’s disease with PET ligand APN-1607102
Plasma biomarkers for Alzheimer’s Disease in relation to neuropathology and cognitive change97
The olfactory nerve is not a likely route to brain infection in COVID-19: a critical review of data from humans and animal models94
Microglia and monocytes in inflammatory CNS disease: integrating phenotype and function90
Tau strains shape disease86
Diverse human astrocyte and microglial transcriptional responses to Alzheimer’s pathology83
Brain arteriolosclerosis81
α-Synuclein in blood exosomes immunoprecipitated using neuronal and oligodendroglial markers distinguishes Parkinson’s disease from multiple system atrophy81
Mitochondrial bioenergetic deficits in C9orf72 amyotrophic lateral sclerosis motor neurons cause dysfunctional axonal homeostasis76
Microvascular injury and hypoxic damage: emerging neuropathological signatures in COVID-1976
The mechanistic link between selective vulnerability of the locus coeruleus and neurodegeneration in Alzheimer’s disease75
Patient-derived organoids and orthotopic xenografts of primary and recurrent gliomas represent relevant patient avatars for precision oncology74
Frequency of LATE neuropathologic change across the spectrum of Alzheimer’s disease neuropathology: combined data from 13 community-based or population-based autopsy cohorts71
Lesion stage-dependent causes for impaired remyelination in MS71
Large-scale pathway specific polygenic risk and transcriptomic community network analysis identifies novel functional pathways in Parkinson disease68
Exosomes induce endolysosomal permeabilization as a gateway by which exosomal tau seeds escape into the cytosol68
The subcellular arrangement of alpha-synuclein proteoforms in the Parkinson’s disease brain as revealed by multicolor STED microscopy68
Analyzing microglial phenotypes across neuropathologies: a practical guide66
The role of hnRNPs in frontotemporal dementia and amyotrophic lateral sclerosis63
Characterizing tau deposition in chronic traumatic encephalopathy (CTE): utility of the McKee CTE staging scheme59
Inhibition of Bruton’s tyrosine kinase interferes with pathogenic B-cell development in inflammatory CNS demyelinating disease58
TERT promoter mutation status is necessary and sufficient to diagnose IDH-wildtype diffuse astrocytic glioma with molecular features of glioblastoma57
Extrinsic immune cell-derived, but not intrinsic oligodendroglial factors contribute to oligodendroglial differentiation block in multiple sclerosis56
Multiple system atrophy-associated oligodendroglial protein p25α stimulates formation of novel α-synuclein strain with enhanced neurodegenerative potential54
Neuron-specific activation of necroptosis signaling in multiple sclerosis cortical grey matter53
Evidence of distinct α-synuclein strains underlying disease heterogeneity53
Primary mismatch repair deficient IDH-mutant astrocytoma (PMMRDIA) is a distinct type with a poor prognosis51
Physiological and pathological functions of TMEM106B: a gene associated with brain aging and multiple brain disorders50
TDP-43 transports ribosomal protein mRNA to regulate axonal local translation in neuronal axons50
Perivascular space dilation is associated with vascular amyloid-β accumulation in the overlying cortex49
Atypical teratoid/rhabdoid tumors (ATRTs) with SMARCA4 mutation are molecularly distinct from SMARCB1-deficient cases48
Interleukin-1 promotes autoimmune neuroinflammation by suppressing endothelial heme oxygenase-1 at the blood–brain barrier48
White matter microglia heterogeneity in the CNS48
Enhanced axonal response of mitochondria to demyelination offers neuroprotection: implications for multiple sclerosis48
Frontal white matter lesions in Alzheimer’s disease are associated with both small vessel disease and AD-associated cortical pathology48
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