Acta Neuropathologica

Papers
(The median citation count of Acta Neuropathologica is 8. The table below lists those papers that are above that threshold based on CrossRef citation counts [max. 250 papers]. The publications cover those that have been published in the past four years, i.e., from 2020-05-01 to 2024-05-01.)
ArticleCitations
Neuropathology of COVID-19: a spectrum of vascular and acute disseminated encephalomyelitis (ADEM)-like pathology384
Plasma p-tau231: a new biomarker for incipient Alzheimer’s disease pathology292
The physiological roles of tau and Aβ: implications for Alzheimer’s disease pathology and therapeutics225
Distribution patterns of tau pathology in progressive supranuclear palsy219
Plasma p-tau181 accurately predicts Alzheimer’s disease pathology at least 8 years prior to post-mortem and improves the clinical characterisation of cognitive decline212
TREM2 activation on microglia promotes myelin debris clearance and remyelination in a model of multiple sclerosis185
Distinct amyloid-β and tau-associated microglia profiles in Alzheimer’s disease167
Cerebral blood flow decrease as an early pathological mechanism in Alzheimer's disease154
Periphery and brain, innate and adaptive immunity in Parkinson’s disease140
CDKN2A/B homozygous deletion is associated with early recurrence in meningiomas120
APOE and TREM2 regulate amyloid-responsive microglia in Alzheimer’s disease118
Correlates of critical illness-related encephalopathy predominate postmortem COVID-19 neuropathology114
Accumulation of amyloid precursor protein C-terminal fragments triggers mitochondrial structure, function, and mitophagy defects in Alzheimer’s disease models and human brains113
Neuropathological consensus criteria for the evaluation of Lewy pathology in post-mortem brains: a multi-centre study109
Cryo-EM structures of tau filaments from Alzheimer’s disease with PET ligand APN-1607102
Plasma biomarkers for Alzheimer’s Disease in relation to neuropathology and cognitive change97
The olfactory nerve is not a likely route to brain infection in COVID-19: a critical review of data from humans and animal models94
Microglia and monocytes in inflammatory CNS disease: integrating phenotype and function90
Tau strains shape disease86
Diverse human astrocyte and microglial transcriptional responses to Alzheimer’s pathology83
α-Synuclein in blood exosomes immunoprecipitated using neuronal and oligodendroglial markers distinguishes Parkinson’s disease from multiple system atrophy81
Brain arteriolosclerosis81
Microvascular injury and hypoxic damage: emerging neuropathological signatures in COVID-1976
Mitochondrial bioenergetic deficits in C9orf72 amyotrophic lateral sclerosis motor neurons cause dysfunctional axonal homeostasis76
The mechanistic link between selective vulnerability of the locus coeruleus and neurodegeneration in Alzheimer’s disease75
Patient-derived organoids and orthotopic xenografts of primary and recurrent gliomas represent relevant patient avatars for precision oncology74
Lesion stage-dependent causes for impaired remyelination in MS71
Frequency of LATE neuropathologic change across the spectrum of Alzheimer’s disease neuropathology: combined data from 13 community-based or population-based autopsy cohorts71
The subcellular arrangement of alpha-synuclein proteoforms in the Parkinson’s disease brain as revealed by multicolor STED microscopy68
Large-scale pathway specific polygenic risk and transcriptomic community network analysis identifies novel functional pathways in Parkinson disease68
Exosomes induce endolysosomal permeabilization as a gateway by which exosomal tau seeds escape into the cytosol68
Analyzing microglial phenotypes across neuropathologies: a practical guide66
The role of hnRNPs in frontotemporal dementia and amyotrophic lateral sclerosis63
Characterizing tau deposition in chronic traumatic encephalopathy (CTE): utility of the McKee CTE staging scheme59
Inhibition of Bruton’s tyrosine kinase interferes with pathogenic B-cell development in inflammatory CNS demyelinating disease58
TERT promoter mutation status is necessary and sufficient to diagnose IDH-wildtype diffuse astrocytic glioma with molecular features of glioblastoma57
Extrinsic immune cell-derived, but not intrinsic oligodendroglial factors contribute to oligodendroglial differentiation block in multiple sclerosis56
Multiple system atrophy-associated oligodendroglial protein p25α stimulates formation of novel α-synuclein strain with enhanced neurodegenerative potential54
Evidence of distinct α-synuclein strains underlying disease heterogeneity53
Neuron-specific activation of necroptosis signaling in multiple sclerosis cortical grey matter53
Primary mismatch repair deficient IDH-mutant astrocytoma (PMMRDIA) is a distinct type with a poor prognosis51
TDP-43 transports ribosomal protein mRNA to regulate axonal local translation in neuronal axons50
Physiological and pathological functions of TMEM106B: a gene associated with brain aging and multiple brain disorders50
Perivascular space dilation is associated with vascular amyloid-β accumulation in the overlying cortex49
Frontal white matter lesions in Alzheimer’s disease are associated with both small vessel disease and AD-associated cortical pathology48
Atypical teratoid/rhabdoid tumors (ATRTs) with SMARCA4 mutation are molecularly distinct from SMARCB1-deficient cases48
Interleukin-1 promotes autoimmune neuroinflammation by suppressing endothelial heme oxygenase-1 at the blood–brain barrier48
White matter microglia heterogeneity in the CNS48
Enhanced axonal response of mitochondria to demyelination offers neuroprotection: implications for multiple sclerosis48
Lewy pathology of the esophagus correlates with the progression of Lewy body disease: a Japanese cohort study of autopsy cases47
Structure of Tau filaments in Prion protein amyloidoses47
Patient-derived orthotopic xenografts of pediatric brain tumors: a St. Jude resource46
R1441G but not G2019S mutation enhances LRRK2 mediated Rab10 phosphorylation in human peripheral blood neutrophils46
Pyroptosis in Alzheimer’s disease: cell type-specific activation in microglia, astrocytes and neurons46
SFPQ and Tau: critical factors contributing to rapid progression of Alzheimer’s disease46
Meningeal inflammation in multiple sclerosis induces phenotypic changes in cortical microglia that differentially associate with neurodegeneration46
Congenic expression of poly-GA but not poly-PR in mice triggers selective neuron loss and interferon responses found in C9orf72 ALS45
Infratentorial IDH-mutant astrocytoma is a distinct subtype45
Clinical and molecular heterogeneity of pineal parenchymal tumors: a consensus study45
LATE-NC staging in routine neuropathologic diagnosis: an update45
The coarse-grained plaque: a divergent Aβ plaque-type in early-onset Alzheimer’s disease45
Insulin-like growth factor 2 (IGF2) protects against Huntington’s disease through the extracellular disposal of protein aggregates44
ETMR: a tumor entity in its infancy43
C9orf72 loss-of-function: a trivial, stand-alone or additive mechanism in C9 ALS/FTD?40
Profiling the neurovascular unit unveils detrimental effects of osteopontin on the blood–brain barrier in acute ischemic stroke40
Selective vulnerability of inhibitory networks in multiple sclerosis40
Complement component 3 from astrocytes mediates retinal ganglion cell loss during neuroinflammation39
Supratentorial ependymoma in childhood: more than just RELA or YAP38
Genome-wide meta-analysis for Alzheimer’s disease cerebrospinal fluid biomarkers38
Recurrent fusions in PLAGL1 define a distinct subset of pediatric-type supratentorial neuroepithelial tumors37
Integrated molecular and clinical analysis of low-grade gliomas in children with neurofibromatosis type 1 (NF1)36
Rainwater Charitable Foundation criteria for the neuropathologic diagnosis of progressive supranuclear palsy36
Neuronal activity modulates alpha-synuclein aggregation and spreading in organotypic brain slice cultures and in vivo36
PATZ1 fusions define a novel molecularly distinct neuroepithelial tumor entity with a broad histological spectrum36
The existence of Aβ strains and their potential for driving phenotypic heterogeneity in Alzheimer’s disease35
Maturation of neuronal AD-tau pathology involves site-specific phosphorylation of cytoplasmic and synaptic tau preceding conformational change and fibril formation35
Post-mortem analyses of PiB and flutemetamol in diffuse and cored amyloid-β plaques in Alzheimer’s disease35
Making sense of missense variants in TTN-related congenital myopathies35
Subgroup and subtype-specific outcomes in adult medulloblastoma35
Distinct tau neuropathology and cellular profiles of an APOE3 Christchurch homozygote protected against autosomal dominant Alzheimer’s dementia35
Upstream open reading frame with NOTCH2NLC GGC expansion generates polyglycine aggregates and disrupts nucleocytoplasmic transport: implications for polyglycine diseases35
Age-associated insolubility of parkin in human midbrain is linked to redox balance and sequestration of reactive dopamine metabolites34
Cryo-EM structures of prion protein filaments from Gerstmann–Sträussler–Scheinker disease34
A multifactorial model of pathology for age of onset heterogeneity in familial Alzheimer’s disease33
Chronic traumatic encephalopathy (CTE): criteria for neuropathological diagnosis and relationship to repetitive head impacts33
In vitro amplification of pathogenic tau conserves disease-specific bioactive characteristics32
BTK inhibition limits B-cell–T-cell interaction through modulation of B-cell metabolism: implications for multiple sclerosis therapy32
Non-IDH1-R132H IDH1/2 mutations are associated with increased DNA methylation and improved survival in astrocytomas, compared to IDH1-R132H mutations32
Seizure-mediated iron accumulation and dysregulated iron metabolism after status epilepticus and in temporal lobe epilepsy32
Frequent inactivating mutations of the PBAF complex gene PBRM1 in meningioma with papillary features32
Clear cell meningiomas are defined by a highly distinct DNA methylation profile and mutations in SMARCE131
Lipids, lysosomes and mitochondria: insights into Lewy body formation from rare monogenic disorders31
Complement-associated loss of CA2 inhibitory synapses in the demyelinated hippocampus impairs memory31
CDKN2A deletion in supratentorial ependymoma with RELA alteration indicates a dismal prognosis: a retrospective analysis of the HIT ependymoma trial cohort31
Cross-platform transcriptional profiling identifies common and distinct molecular pathologies in Lewy body diseases31
Altered ceramide metabolism is a feature in the extracellular vesicle-mediated spread of alpha-synuclein in Lewy body disorders30
The proteome of granulovacuolar degeneration and neurofibrillary tangles in Alzheimer’s disease29
Distinct characteristics of limbic-predominant age-related TDP-43 encephalopathy in Lewy body disease29
Prion propagation estimated from brain diffusion MRI is subtype dependent in sporadic Creutzfeldt–Jakob disease28
Multi-omic molecular profiling reveals potentially targetable abnormalities shared across multiple histologies of brain metastasis28
Vesicle trafficking and lipid metabolism in synucleinopathy28
Distinct clinicopathologic clusters of persons with TDP-43 proteinopathy28
Mosaic trisomy of chromosome 1q in human brain tissue associates with unilateral polymicrogyria, very early-onset focal epilepsy, and severe developmental delay28
Malignant transformation of a polymorphous low grade neuroepithelial tumor of the young (PLNTY)27
Cryo-EM structures of amyloid-β filaments with the Arctic mutation (E22G) from human and mouse brains27
Single-cell profiling of myasthenia gravis identifies a pathogenic T cell signature27
Altered oligodendroglia and astroglia in chronic traumatic encephalopathy27
Glioblastomas with primitive neuronal component harbor a distinct methylation and copy-number profile with inactivation of TP53, PTEN, and RB126
Necrotic reshaping of the glioma microenvironment drives disease progression26
Tau immunotherapy is associated with glial responses in FTLD-tau26
Accumulation of TMEM106B C-terminal fragments in neurodegenerative disease and aging26
Ultrastructural and biochemical classification of pathogenic tau, α-synuclein and TDP-4326
Apolipoprotein E regulates lipid metabolism and α-synuclein pathology in human iPSC-derived cerebral organoids25
APOE4 exacerbates α-synuclein seeding activity and contributes to neurotoxicity in Alzheimer’s disease with Lewy body pathology25
CSF p-tau increase in response to Aβ-type and Danish-type cerebral amyloidosis and in the absence of neurofibrillary tangles25
HnRNP K mislocalisation is a novel protein pathology of frontotemporal lobar degeneration and ageing and leads to cryptic splicing25
Oligodendroglia heterogeneity in the human central nervous system24
Epigenomic, genomic, and transcriptomic landscape of schwannomatosis24
ApoE4 inhibition of VMAT2 in the locus coeruleus exacerbates Tau pathology in Alzheimer’s disease24
Heterogeneity in α-synuclein fibril activity correlates to disease phenotypes in Lewy body dementia24
HIF-1α is involved in blood–brain barrier dysfunction and paracellular migration of bacteria in pneumococcal meningitis24
Phenotypic diversity of genetic Creutzfeldt–Jakob disease: a histo-molecular-based classification24
Targeting cancer stem cells in medulloblastoma by inhibiting AMBRA1 dual function in autophagy and STAT3 signalling24
Spatiotemporal characterization of cellular tau pathology in the human locus coeruleus–pericoerulear complex by three-dimensional imaging23
Developmental malformations in Huntington disease: neuropathologic evidence of focal neuronal migration defects in a subset of adult brains23
Impairment of the mitochondrial one-carbon metabolism enzyme SHMT2 causes a novel brain and heart developmental syndrome23
Reduction of advanced tau-mediated memory deficits by the MAP kinase p38γ23
Patterns of amygdala region pathology in LATE-NC: subtypes that differ with regard to TDP-43 histopathology, genetic risk factors, and comorbid pathologies23
Trafficking of the glutamate transporter is impaired in LRRK2-related Parkinson’s disease23
The oncogenic fusion landscape in pediatric CNS neoplasms23
miR155 regulation of behavior, neuropathology, and cortical transcriptomics in Alzheimer's disease22
Wolframin is a novel regulator of tau pathology and neurodegeneration22
Retinal pathological features and proteome signatures of Alzheimer’s disease22
MOG-expressing teratoma followed by MOG-IgG-positive optic neuritis21
Heterogeneity of white matter astrocytes in the human brain21
Mutations within FGFR1 are associated with superior outcome in a series of 83 diffuse midline gliomas with H3F3A K27M mutations21
Antibody against TDP-43 phosphorylated at serine 375 suggests conformational differences of TDP-43 aggregates among FTLD–TDP subtypes21
DDX17 is involved in DNA damage repair and modifies FUS toxicity in an RGG-domain dependent manner20
Vagus nerve inflammation contributes to dysautonomia in COVID-1920
Increased pyroptosis activation in white matter microglia is associated with neuronal loss in ALS motor cortex20
Association of small vessel disease with tau pathology20
Physiological β-amyloid clearance by the liver and its therapeutic potential for Alzheimer’s disease20
FMNL2 regulates gliovascular interactions and is associated with vascular risk factors and cerebrovascular pathology in Alzheimer’s disease20
Multiomic elucidation of a coding 99-mer repeat-expansion skeletal muscle disease20
Molecular characterization of CNS paragangliomas identifies cauda equina paragangliomas as a distinct tumor entity20
Germline-driven replication repair-deficient high-grade gliomas exhibit unique hypomethylation patterns20
Rapid-CNS2: rapid comprehensive adaptive nanopore-sequencing of CNS tumors, a proof-of-concept study20
TDP-43 drives synaptic and cognitive deterioration following traumatic brain injury20
A new non-aggregative splicing isoform of human Tau is decreased in Alzheimer’s disease20
The KBTBD6/7-DRD2 axis regulates pituitary adenoma sensitivity to dopamine agonist treatment19
Galectin-3 is elevated in CSF and is associated with Aβ deposits and tau aggregates in brain tissue in Alzheimer’s disease19
Association of probable REM sleep behavior disorder with pathology and years of contact sports play in chronic traumatic encephalopathy19
Skeletal muscle provides the immunological micro-milieu for specific plasma cells in anti-synthetase syndrome-associated myositis19
Systemic sclerosis-associated myositis features minimal inflammation and characteristic capillary pathology19
Limbic-predominant age-related TDP-43 encephalopathy neuropathological change (LATE-NC) is independently associated with dementia and strongly associated with arteriolosclerosis in the oldest-old19
Transmissible α-synuclein seeding activity in brain and stomach of patients with Parkinson’s disease19
Alzheimer disease neuropathology in a patient previously treated with aducanumab19
TDP-43 interacts with pathological τ protein in Alzheimer’s disease19
Oligosarcomas, IDH-mutant are distinct and aggressive19
Transcriptomic analysis of frontotemporal lobar degeneration with TDP-43 pathology reveals cellular alterations across multiple brain regions19
Alzheimer’s disease neuropathological change three decades after iatrogenic amyloid-β transmission18
TERT promoter mutation and chromosome 6 loss define a high-risk subtype of ependymoma evolving from posterior fossa subependymoma18
Genome-wide association study and functional validation implicates JADE1 in tauopathy18
TREM2 expression in the brain and biological fluids in prion diseases18
DNA methylation-based classification of malformations of cortical development in the human brain18
Neuronal spreading and plaque induction of intracellular Aβ and its disruption of Aβ homeostasis18
Immunisation with UB-312 in the Thy1SNCA mouse prevents motor performance deficits and oligomeric α-synuclein accumulation in the brain and gut17
Transmission of cervid prions to humanized mice demonstrates the zoonotic potential of CWD17
Adaptive structural changes in the motor cortex and white matter in Parkinson’s disease17
Intellectual disability: dendritic anomalies and emerging genetic perspectives17
An interaction between synapsin and C9orf72 regulates excitatory synapses and is impaired in ALS/FTD17
Altered DNA methylation profiles in blood from patients with sporadic Creutzfeldt–Jakob disease17
EZHIP: a new piece of the puzzle towards understanding pediatric posterior fossa ependymoma17
PSD-93 up-regulates the synaptic activity of corticotropin-releasing hormone neurons in the paraventricular nucleus in depression16
LRP10 interacts with SORL1 in the intracellular vesicle trafficking pathway in non-neuronal brain cells and localises to Lewy bodies in Parkinson’s disease and dementia with Lewy bodies16
Wide distribution of prion infectivity in the peripheral tissues of vCJD and sCJD patients16
Low-grade glioneuronal tumors with FGFR2 fusion resolve into a single epigenetic group corresponding to ‘Polymorphous low-grade neuroepithelial tumor of the young’16
GOPC:ROS1 and other ROS1 fusions represent a rare but recurrent drug target in a variety of glioma types16
Degeneration of the locus coeruleus is a common feature of tauopathies and distinct from TDP-43 proteinopathies in the frontotemporal lobar degeneration spectrum16
Cross-regional homeostatic and reactive glial signatures in multiple sclerosis16
Biomarkers for parkinsonian disorders in CNS-originating EVs: promise and challenges16
Signature laminar distributions of pathology in frontotemporal lobar degeneration15
Evidence of disrupted rhombic lip development in the pathogenesis of Dandy–Walker malformation15
Consequences of variability in α-synuclein fibril structure on strain biology15
A novel ATXN1-DUX4 fusion expands the spectrum of ‘CIC-rearranged sarcoma’ of the CNS to include non-CIC alterations15
Comparing amyloid-β plaque burden with antemortem PiB PET in autosomal dominant and late-onset Alzheimer disease15
Frequency and distribution of TAR DNA-binding protein 43 (TDP-43) pathology increase linearly with age in a large cohort of older adults with and without dementia15
ATRT–SHH comprises three molecular subgroups with characteristic clinical and histopathological features and prognostic significance15
Loss of LAMP5 interneurons drives neuronal network dysfunction in Alzheimer’s disease15
Aminopeptidase A contributes to biochemical, anatomical and cognitive defects in Alzheimer’s disease (AD) mouse model and is increased at early stage in sporadic AD brain15
TMEM106B modifies TDP-43 pathology in human ALS brain and cell-based models of TDP-43 proteinopathy15
Antibodies to MOG in CSF only: pathological findings support the diagnostic value15
Genetics of synucleins in neurodegenerative diseases15
The immunohistochemical, DNA methylation, and chromosomal copy number profile of cauda equina paraganglioma is distinct from extra-spinal paraganglioma15
Targeting fibroblast growth factor receptors to combat aggressive ependymoma15
Selective vulnerability of tripartite synapses in amyotrophic lateral sclerosis14
Identification of TMEM106B amyloid fibrils provides an updated view of TMEM106B biology in health and disease14
The proteomic landscape of glioblastoma recurrence reveals novel and targetable immunoregulatory drivers14
Environmental and host factors that contribute to prion strain evolution14
Brain region-specific susceptibility of Lewy body pathology in synucleinopathies is governed by α-synuclein conformations14
Dominant-acting CSF1R variants cause microglial depletion and altered astrocytic phenotype in zebrafish and adult-onset leukodystrophy14
Photodynamic studies reveal rapid formation and appreciable turnover of tau inclusions14
Oncostatin M triggers brain inflammation by compromising blood–brain barrier integrity14
ATP10B and the risk for Parkinson’s disease14
Molecular profiling of pediatric meningiomas shows tumor characteristics distinct from adult meningiomas13
Enhancer reprogramming in PRC2-deficient malignant peripheral nerve sheath tumors induces a targetable de-differentiated state13
Genetic alterations of TP53 and OTX2 indicate increased risk of relapse in WNT medulloblastomas13
A network of core and subtype-specific gene expression programs in myositis13
High-grade glioma with pleomorphic and pseudopapillary features (HPAP): a proposed type of circumscribed glioma in adults harboring frequent TP53 mutations and recurrent monosomy 1313
Early white matter pathology in the fornix of the limbic system in Huntington disease13
Multiplatform molecular analyses refine classification of gliomas arising in patients with neurofibromatosis type 113
Repetitive head impacts and chronic traumatic encephalopathy are associated with TDP-43 inclusions and hippocampal sclerosis13
Identification of retinoblastoma binding protein 7 (Rbbp7) as a mediator against tau acetylation and subsequent neuronal loss in Alzheimer’s disease and related tauopathies13
Phosphorylated tau in the retina correlates with tau pathology in the brain in Alzheimer’s disease and primary tauopathies13
A glutaminyl cyclase-catalyzed α-synuclein modification identified in human synucleinopathies13
Functional excitatory to inhibitory synaptic imbalance and loss of cognitive performance in people with Alzheimer’s disease neuropathologic change12
Infratentorial C11orf95-fused gliomas share histologic, immunophenotypic, and molecular characteristics of supratentorial RELA-fused ependymoma12
Neurofibromatosis type 2 predisposes to ependymomas of various localization, histology, and molecular subtype12
Huntington’s disease brain-derived small RNAs recapitulate associated neuropathology in mice12
Unique seeding profiles and prion-like propagation of synucleinopathies are highly dependent on the host in human α-synuclein transgenic mice12
Histopathology of the cerebellar cortex in essential tremor and other neurodegenerative motor disorders: comparative analysis of 320 brains12
Nuclear depletion of RNA-binding protein ELAVL3 (HuC) in sporadic and familial amyotrophic lateral sclerosis12
Circular RNA profiling distinguishes medulloblastoma groups and shows aberrant RMST overexpression in WNT medulloblastoma12
Molecular characterization of DICER1-mutated pituitary blastoma12
α-Synuclein molecular behavior and nigral proteomic profiling distinguish subtypes of Lewy body disorders12
Heteroplasmic mitochondrial DNA mutations in frontotemporal lobar degeneration12
More than meets the eye in Parkinson’s disease and other synucleinopathies: from proteinopathy to lipidopathy11
Cryptic exon detection and transcriptomic changes revealed in single-nuclei RNA sequencing of C9ORF72 patients spanning the ALS-FTD spectrum11
Mutant LRRK2 exacerbates immune response and neurodegeneration in a chronic model of experimental colitis11
Receptor clustering and pathogenic complement activation in myasthenia gravis depend on synergy between antibodies with multiple subunit specificities11
C11orf95-RELA reprograms 3D epigenome in supratentorial ependymoma11
DNA methylation analysis of glioblastomas harboring FGFR3-TACC3 fusions identifies a methylation subclass with better patient survival11
Expanded analysis of high-grade astrocytoma with piloid features identifies an epigenetically and clinically distinct subtype associated with neurofibromatosis type 111
Dysregulated coordination of MAPT exon 2 and exon 10 splicing underlies different tau pathologies in PSP and AD11
EWSR1-BEND2 fusion defines an epigenetically distinct subtype of astroblastoma11
Stress-inducible phosphoprotein 1 (HOP/STI1/STIP1) regulates the accumulation and toxicity of α-synuclein in vivo11
Neuropathological associations of limbic-predominant age-related TDP-43 encephalopathy neuropathological change (LATE-NC) differ between the oldest-old and younger-old11
TREM2 gene expression associations with Alzheimer’s disease neuropathology are region-specific: implications for cortical versus subcortical microglia11
Concussion leads to widespread axonal sodium channel loss and disruption of the node of Ranvier11
Aberrant NOVA1 function disrupts alternative splicing in early stages of amyotrophic lateral sclerosis11
Brain pathologies are associated with both the rate and variability of declining motor function in older adults11
Neuropathologic scales of cerebrovascular disease associated with diffusion changes on MRI11
Donanemab detects a minor fraction of amyloid-β plaques in post-mortem brain tissue of patients with Alzheimer’s disease and Down syndrome11
Archeological neuroimmunology: resurrection of a pathogenic immune response from a historical case sheds light on human autoimmune encephalomyelitis and multiple sclerosis10
Clinical and neuropathological diversity of tauopathy in MAPT duplication carriers10
Associations of psychiatric disease and ageing with FKBP5 expression converge on superficial layer neurons of the neocortex10
Oncohistone interactome profiling uncovers contrasting oncogenic mechanisms and identifies potential therapeutic targets in high grade glioma10
Amplification of the PLAG-family genes—PLAGL1 and PLAGL2—is a key feature of the novel tumor type CNS embryonal tumor with PLAGL amplification10
New SNCA mutation and structures of α-synuclein filaments from juvenile-onset synucleinopathy10
Intratumor and informatic heterogeneity influence meningioma molecular classification10
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