Brain & Development

Papers
(The H4-Index of Brain & Development is 12. The table below lists those papers that are above that threshold based on CrossRef citation counts [max. 250 papers]. The publications cover those that have been published in the past four years, i.e., from 2021-11-01 to 2025-11-01.)
ArticleCitations
Differentiating early clinical features of Panayiotopoulos syndrome from acute encephalopathy27
Intellectual disability and genotype-phenotype correlation between full-scale intelligence quotient and mutation characteristics in boys with dystrophinopathy23
Autosomal dominant ADAR c.3019G>A (p.(G1007R)) variant is an important mimic of hereditary spastic paraplegia and cerebral palsy23
New Year’s greetings20
Tendency and risk factors of acute pancreatitis in children with severe motor and intellectual disabilities: A single-center study19
Acknowledgments to Anonymous Reviewers in 202317
Current state of hemispherectomy and callosotomy for pediatric refractory epilepsy in Denmark17
Next generation sequencing in children with unexplained epilepsy: A retrospective cohort study16
Association of O blood type with the prognosis of acute necrotizing encephalopathy in childhood: A single-center cohort study16
A case of spinal muscular atrophy type 0 treated with nusinersen without progression of early-onset scoliosis – possibility of preventing scoliosis with a rehabilitation program focusing on postural m13
Interacting with AP1 complex mutated synergin gamma (SYNRG) reveals a novel coatopathy in the form of complicated hereditary spastic paraplegia13
Potential of arterial spin labeling in elucidating the pathogenesis of the splenium of the corpus callosum and cerebellar dentate nucleus in encephalopathy13
Cover12
Refractory status epilepticus with fever due to mumps vaccine-induced encephalitis caused secondary encephalopathy mimicking acute encephalopathy with biphasic seizures and late reduced diffusion12
Video game exposure in children with epilepsy: EEG and clinical findings12
The first case of Al-Raqad syndrome in Japan is associated with a homozygous DCPS exonic variant resulting in aberrant splicing12
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