OOIR: Observatory of International Research

Papers

(The H4-Index of Mucosal Immunology is 38. The table below lists those papers that are above that threshold based on CrossRef citation counts [max. 250 papers]. The publications cover those that have been published in the past four years, i.e., from 2020-05-01 to 2024-05-01.)

Article	Citations
Perturbation of the gut microbiome by Prevotella spp. enhances host susceptibility to mucosal inflammation	216
The lung–gut axis during viral respiratory infections: the impact of gut dysbiosis on secondary disease outcomes	167
The central role of the nasal microenvironment in the transmission, modulation, and clinical progression of SARS-CoV-2 infection	166
Human gut-associated lymphoid tissues (GALT); diversity, structure, and function	160
Systemic and mucosal IgA responses are variably induced in response to SARS-CoV-2 mRNA vaccination and are associated with protection against subsequent infection	148
Animal and translational models of SARS-CoV-2 infection and COVID-19	143
The intestinal neuro-immune axis: crosstalk between neurons, immune cells, and microbes	133
Cellular and functional heterogeneity of the airway epithelium	119
Introduction to host microbiome symbiosis in health and disease	96
Intestinal epithelial NAIP/NLRC4 restricts systemic dissemination of the adapted pathogen Salmonella Typhimurium due to site-specific bacterial PAMP expression	91
The impact of the lung environment on macrophage development, activation and function: diversity in the face of adversity	86
Dietary fiber metabolites regulate innate lymphoid cell responses	81
The NLRP6 inflammasome in health and disease	79
Tissue resident memory T cells in the respiratory tract	78
MAIT cells, guardians of skin and mucosa?	65
Long-term maintenance of lung resident memory T cells is mediated by persistent antigen	63
Neutrophils in respiratory viral infections	63
IL-17 production by tissue-resident MAIT cells is locally induced in children with pneumonia	62
Crosstalk between the oral microbiota, mucosal immunity, and the epithelial barrier regulates oral mucosal disease pathogenesis	55
MAIT cell-directed therapy of Mycobacterium tuberculosis infection	54
Macrophage metabolic reprogramming during chronic lung disease	54
A monoclonal antibody to Siglec-8 suppresses non-allergic airway inflammation and inhibits IgE-independent mast cell activation	53
Human small intestinal infection by SARS-CoV-2 is characterized by a mucosal infiltration with activated CD8+ T cells	52
Seasonal and pandemic influenza: 100 years of progress, still much to learn	52
Host/microbiota interactions in health and diseases—Time for mucosal microbiology!	52

The emerging roles of eosinophils in mucosal homeostasis	52
Targeted deletion of the TSLP receptor reveals cellular mechanisms that promote type 2 airway inflammation	51
Microbiome-based interventions to modulate gut ecology and the immune system	47
Gut-derived short-chain fatty acids modulate skin barrier integrity by promoting keratinocyte metabolism and differentiation	47
Epithelium-autonomous NAIP/NLRC4 prevents TNF-driven inflammatory destruction of the gut epithelial barrier in Salmonella-infected mice	46
The role of miR-155 in cigarette smoke-induced pulmonary inflammation and COPD	45
CD4+ T cells persist for years in the human small intestine and display a TH1 cytokine profile	43
Granulocytes act as a niche for Mycobacterium tuberculosis growth	42
Dealing with a mucosal viral pandemic: lessons from COVID-19 vaccines	42
Enteric neuroimmune interactions coordinate intestinal responses in health and disease	42
IL-22 receptor signaling in Paneth cells is critical for their maturation, microbiota colonization, Th17-related immune responses, and anti-Salmonella immunity	41
Intestinal epithelial cell metabolism at the interface of microbial dysbiosis and tissue injury	39
Host responses to mucosal biofilms in the lung and gut	39
IL-17A both initiates, via IFNγ suppression, and limits the pulmonary type-2 immune response to nematode infection	38
Uric acid pathway activation during respiratory virus infection promotes Th2 immune response via innate cytokine production and ILC2 accumulation	38