Clinical & Translational Immunology

Papers
(The H4-Index of Clinical & Translational Immunology is 21. The table below lists those papers that are above that threshold based on CrossRef citation counts [max. 250 papers]. The publications cover those that have been published in the past four years, i.e., from 2022-01-01 to 2026-01-01.)
ArticleCitations
107
92
SARS‐CoV‐2‐associated T‐cell infiltration in the central nervous system52
The spectrum of B cells in the pathogenesis, diagnosis and therapeutic applications of immunoglobulin G4‐related disease43
Circulating cytokine and chemokine patterns associated with cytomegalovirus reactivation after stem cell transplantation39
Characteristics of PD‐1+CD4+ T cells in peripheral blood and synovium of rheumatoid arthritis patients37
Biomarkers to predict and diagnose pulmonary complications in children post haematopoietic stem cell transplant36
Polyfunctional T peripheral helper cells are associated with the magnitude and durability of antibody responses after COVID‐1931
An optimised whole blood interleukin‐2 release assay is more sensitive than interferon‐γ ELIS pot for detecting and quantifying gluten‐specific 27
Impact of adaptive natural killer cells, KLRC2 genotype and cytomegalovirus reactivation on late mortality in patients with severe COVID‐19 lung disease27
26
Effects of extremely preterm birth on cytokine and chemokine responses induced by T‐cell activation during infancy25
Heterologous booster vaccination with CoronaVac following prime vaccination with mRNA vaccine25
25
Retrospective analysis of the efficacy of adjuvant cytokine‐induced killer cell immunotherapy combined with chemotherapy in colorectal cancer patients after surgery24
24
23
Targeting BMI‐1 to deplete antibody‐secreting cells in autoimmunity22
Endogenous bystander killing mechanisms enhance the activity of novel FAP‐specific CAR‐T cells against glioblastoma21
Adeno‐associated viral vectors encoding anti‐P2X7 nanobodies reduce graft‐versus‐host disease in a humanised mouse model21
The contribution of the alternative pathway in complement activation on cell surfaces depends on the strength of classical pathway initiation21
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